DR MANISH- ppt Laser Proctology piles & Fistula

Jan 27, 2025Download as PPTX, PDF0 likes4 views

Get the best piles and fistula treatment in Jaipur with advanced laser proctology techniques. Dr. Manish Rajput, a renowned specialist, provides painless, quick, and highly effective treatments for piles, fissures, and fistula. Using cutting-edge laser technology, the procedures ensure minimal discomfort, no stitches, and faster recovery, helping patients return to their daily lives with ease. Dr. Rajput is committed to offering personalized care and long-term solutions, ensuring lasting relief and improved quality of life. With his expertise and modern facilities, you can expect safe and reliable treatment for all proctological conditions.

D R
M A N I S H
RAJPUT
ht tps://drmanishrajput.com
Book an appointme
nt!

IN T R O D U C T IO
N
Dr
. Manish Rajput is an
Interventional Radiologist & Team
Lead, Team IR Jaipur
. They are the
biggest team of Interventional
Radiologists. They are trained from
Tata Memorial Center, Mumbai,
India. They have worked in so
many government and corporate
hospitals across the country.

Medical school (MBBS): 2005-2011: -
People’s Medical College, Bhopal(MP)
DNB (Radio diagnosis):- Apollo
hospital, Hyderabad(Telangana)
FVIR (PDCC): - Tata Memorial
Centre, Mumbai(Maharashtra)
Senior Resident: Hinduja Hospital Mumbai,
SMS Hospital Jaipur
Past Visiting Doctor: Leelavati Hospital
Mumbai, Breach Candy Hospital Mumbai,
Wockhardt Hospital Mumbai, Hinduja
Hospital Mumbai
Ex Assitant Professor: JNU Medical College,
Jaipur Currently Working as Senior Consultant
Interventional Radiologist in various
corporate hospitals of Rajasthan based in
Jaipur
HIS
EDUCATION

S T R E N G T H S
I lead the biggest IR team in the
state. Vast portfolio for IR
services.
All the team members are from
Tata Memorial Hospital, Mumbai.
Extensive experience in performing
and interpreting basic Radio-
Diagnosis.
Gained experience in performing
Interventional Radiologic
procedures. I possess oratory
skill by speaking at numerous
industry events.
Ability to teach complex concepts in a
basic manner
.

Varicose Vein
s
Prostate Artery
Embolization
PRG
Biopsy an
d fNAC
Angioplasty & Venoplast
y
PCN & DJ Stentin
g
O
U
R
S
E
R
V

+91 7729021111
dr.manish@infinityintervention.com
O-5-A, Adinath Marg, Near Surya
Hospital, C Scheme, Ashok
Nagar, Jaipur, Rajasthan 302001
C ON TA C
T US!

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Comprehensive Management of Peptic Ulcer Disease and GERD I. Introduction Peptic Ulcer Disease (PUD) and Gastroesophageal Reflux Disease (GERD) are distinct yet overlapping disorders of the gastrointestinal system, marked by significant morbidity worldwide. These conditions illustrate the consequence of a disturbed harmony between offensive gastric secretions and the protective barriers of the mucosa. From ancient remedies to modern-day proton pump inhibitors and eradication therapies, the treatment approaches to these disorders represent a triumph of translational medicine. While PUD typically involves mucosal erosion in the stomach or proximal duodenum due to Helicobacter pylori infection or NSAID use, GERD arises from the reflux of gastric contents into the esophagus due to incompetent lower esophageal sphincter tone. Both conditions necessitate a thorough understanding of their etiopathogenesis for rational therapy and long-term management. This document explores the latest, evidence-based treatment paradigms, structured with clarity and clinical relevance. --- II. Peptic Ulcer Disease (PUD) Definition and Epidemiology Peptic ulcers are breaks in the mucosal lining of the stomach or duodenum that penetrate the muscularis mucosa. Gastric ulcers typically occur on the lesser curvature of the stomach, while duodenal ulcers are found in the first part of the duodenum. Globally, the prevalence of PUD has declined, largely due to H. pylori eradication, yet NSAID-related ulcers persist, especially among the elderly. Etiology and Risk Factors Helicobacter pylori infection – Present in ~90% of duodenal and 70% of gastric ulcers. NSAIDs – Inhibit prostaglandin synthesis, compromising mucosal defense. Smoking – Impairs mucosal healing. Stress (critical illness) – Leads to stress ulcers. Zollinger-Ellison Syndrome – Gastrinoma with excess acid secretion. Corticosteroids, alcohol, and genetic predisposition are other contributors. Pathophysiology The balance between aggressive factors (acid, pepsin, H. pylori, NSAIDs) and defensive mechanisms (mucus, bicarbonate, blood flow, prostaglandins) determines mucosal integrity. H. pylori causes chronic inflammation and epithelial damage. NSAIDs decrease prostaglandins, reducing mucosal blood flow and bicarbonate production. --- III. Clinical Features of Peptic Ulcer Epigastric pain: Most common symptom; burning or gnawing in nature. Duodenal ulcers: Pain relieved by food, occurs 2–3 hours after meals. Gastric ulcers: Pain worsens with food intake. Nausea, bloating, early satiety Complications: Bleeding: Hematemesis, melena. Perforation: Sudden severe abdominal pain. Gastric outlet obstruction Penetration into adjacent organs (e.g., pancreas) --- IV. Diagnosis of Peptic Ulcer Endoscopy: Gold standard for diagnosis and biopsy to rule out malignancy. Rapid urease test, histology, urea breath test, stool antigen – for H. pylori. Serologic testing (less preferred). Barium study

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